Essays on Chronic Kidney Disease

Subjective

MN is a 75 y/o African-American female who presents with complaints of shortness of breath, nausea, reduced urine output, fatigue and swellings in the legs as well as the ankles. The patient states that her symptoms began three days prior to her visit to the clinic and has had a decreased urine volume. Initially, the patients attributed the symptoms, especially nausea and fatigue to the side effects of the medications that she used in managing hypertension and diabetes. However, she states that the symptoms have gradually worsened and that was the reason for visiting the clinic. Past medical history: ...

Application– Chronic Kidney Disease

(Author, Department, University,

Chronic Kidney Disease Chronic kidney disease is related to a decrease in kidney function or damage to the kidney function for a minimum of 3 months. Actually, it is an ongoing process in which nephrons’ functioning decrease, glomeruli are destroyed, and glomerular stress is increased as a result of more work by surviving glomeruli to increase glomerular filtration rate (GFR); thereby, resulting in predictable steady decline of the function of kidneys (Buttaro, 2013). Chronic kidney disease is usually difficult to detect or grasp, and symptoms may not appear with a GFR more than 35%. However, when ...

Problem statement for research purpose: Hypertension can be a cause as well as a consequence of chronic kidney disease (CRD). A number of clinical studies have identified the harmful effects of uncontrolled hypertension on the kidney function (Kokubo, 2012; Berlowitz, 2016)). According to NIH statistics, 23.3% individuals without CKD had hypertension in a representative sample. On the other hand, 35.8% of individuals in stage 1, 48.1% in stage 2, 59.9% of stage 3 and 84.1% in stage 4-5 CKD had hypertension (National Institutes of Health, 2010). In stage 4 CKD, the glomerular filtration is the lowest at <15 ...

Why does she have bacteria and white blood cell casts in her urine?

In this case, Ms, Cornwall has focal bacterial infection of her calyces, medulla and renal pelvis. The presence of these bacterial and their invasion within the mentioned parts causes a body response to defend against the invasion. This occurs though a trigger of the inflammatory mediators and other body defense mechanism in this case the white blood cells. With the white blood cells now dominating the region of infection, there occurs a systematic abscess formation and necrosis within the medulla (Alexander et al., 2012). At this point, the next step that could occur is the scar tissue formation which ...